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Any one have any examples of ERP with health ocd / hypochondria?

i am a waste of time is a waste of time try anything around here or wherever i just have a little hope inside that i will get it but i need someone else help me but i am invisible and i dont get it?...A lot of?

I have a hard time adapting to change in the most insignificant things like perfumes accessories or colors when I do my nails. The few times I do I've gotten an urge to go right back to the old thing. Sometimes I can't even sleep because of so much debating in my head ?

I know I shouldn’t, but is there any chance I can get some reassurance or some talking to. I just had the scariest thought and it sent me into panic

Hey so I have a question. Maybe I should already know this but can somebody explain the different types of OCD people talk about on here? Like I've seen HOCD and Pure O and other things like that but what do all of them mean? Thank you

This girl I talk with at school, she’s a friend of a friend... she’s been arguing with her ex boyfriend because she recently discovered she was a lesbian.. and that gave me anxiety and made me overthink and now I’m convinced I’m one too... I tried so hard to avoid it... just today I was getting those good little feelings over a guy in my history... now it feels fake :(

I feel so weird these days. Like I feel blank empty person with no emotions. I mean i still do get anxiety here and there but like no emotions or guilt. I see scary movies and feel no reaction, I imagine hurting other and don’t feel any guilt more like the idgaf typa feeling, before I would be in mental torture crying and depressed about being scared I was a serial killer. I would break down but no I feel urges still by no anxiety or anything. I feel mental and crazy. I would be so excited to wear makeup and excited about impressing guys and dating, but now they don’t seem real to me. Before I would care about OCD and now I’m more like idgaf anymore or what I do. What is going on ?! Am I the only one experiencing this.?

So Im doing Harm EPR. To expose the scariest thing im afraid of. I keep telling my self i would do it again and again and again. And my anxiety sparks. Fear arose. And im just gonna sit with it. Is this normal?

so im suffering from suicidal OCD. its so exhausting just to let them sit in my mind wondering around. it made me depressed. i use to to so afraid, confused, panic and so much anxiety spike. today im feeling numb to all those feelings. its getting into my core believe..there is still resistance thats why im so tired. not 1sec i could distract my self. everywhere i go i feel like its the last place that i will ever visit.

Been a long time since I’ve been on here. But OCD is really kicking my butt tonight. I failed miserably at my exposure hw because my anxiety was too high and had to take an anti anxiety med. I really don’t know how I can keep doing this. My OCD has me convinced I’m a horrible liar and faking medical conditions and if it’s really true, idk, I can’t live with it. Could just use some words of encouragement, anything really...

Methods for dealing with schizpophrenia ocd it seems to be my most prominent one lately... :(

Anyone randomly ever starts talking and midway my brain starts feeling weird so it gets super hard to complete my sentence. And often I’m thinking about something and I feel so deattached like I feel weird af like I can’t describe this weird feeling. Can anyone relate? Also days when I’m feeling good.. I still have the fear like don’t be too happy cause ur OCD will return and torture u randomly. And I keep thinking like I was control myself for a year without harming anyone... but what if I hurt someone randomly and just don’t give a fuck? That’s another fear I have. But yeah please do let me know if your brain feels so weird while ur taking and suddenly it gets hard to talk anymore/ concentrate or even complete it sentence? Please help. I want my old self back again. So So Badly.

does anyone else have mental compulsions? if so, what are they?

Hi great community! i would like u guys to share some experience when doing EPR for harm OCD. like how to do it properly for the first time and what to expect. im going to try my first time soon but a little guidance from u guys are extremely awesome! thank you!

So I’ll preface this by noting how new I am to all this. But what is pure OCD as opposed to just OCD? And what are you experiences with either?

For those who've experienced a decrease in symptoms after practicing ERP- how often did you practice ERP? What kinds of exposures did you do? What else can you suggest to people who are new to treatment?

I’m new here and need help, not as someone who suffers from OCD, but as a mother who’s child (I believe) suffers from OCD. We have not had an official diagnosis yet as I am searching for a physician at this time. She has all of the aspects on the subject specifically, so I really wouldn’t be surprised by an OCD diagnosis at all. My question is, do any of you remember a time when the OCD became a problem for you? I meant, did something happen to trigger it or make it much worse or more evident? I’ve always known she was a “perfectionist” and very intellectually minded, so to speak, but several incidents happened last year (she’s almost 14) with some friends that didn’t end well. Since then, it’s as if she can’t focus at all and gets very obsessed by things. Almost like the incidents last year exacerbated the underlying problem. I want to help her however I can until we can get into a Dr, I just don’t know how and it seems she gets mad and irritated with me when I try, which is hurting this Moms heart. Can any of you suggest what I should or shouldn’t do at this point? She literally becomes frozen with certain fears, like being stung by a bee (never been stung before). Honestly, she’s had a fear of going to the bathroom forever (10 years), but medical Drs have never been able to help and now I know why. So, all to say, I’m not in denial but just want to help her. Thanks for any input you can give.

Hey guys so I suffer from Harm OCD and thoughts about violently harming other and today I’ve been having super minimal thoughts . I had such horrible intrusive thoughts that it feels weird. I’m in constant fear that the thought will come back randomly and the urges will come back stronger than ever. And I’ll actually commit to my urges and act out on them. Like I still Feel weird because even though I don’t have anxiety ... the killer thoughts are in the back of my head. I gueninly don’t dee normal Anymore.

"Prevailing theories indicate that OCD is a biological disease. " Understanding Obsessive-Compulsive and Related Disorders OCD is a common mental disorder, and is often disabling. The past few decades, however, have seen the emergence of many effective treatments, both pharmacological and psychotherapeutic. The challenges for the 21st century are two-fold: first, to make these effective treatments available to all sufferers; and, second, to unravel the biology of this disorder sufficiently so that we can cure its symptoms, and ultimately, prevent its occurrence. In order to tackle these challenges, it is essential that we understand the etiology of OCD. Prevailing theories indicate that OCD is a biological disease. Functional brain imaging studies have produced a model for pathophysiology of OCD which involves hyperactivity in certain subcortical and cortical regions. On the basis of imaging studies, Insel has proposed that inappropriately increased activity in the head of the caudate nucleus inhibits globus pallidus fibers that ordinarily dampen thalamic activity. The resulting increase in thalamic activity produces increased activity in orbitofrontal cortex, which, via the cingulate gyrus, completes the circuit to the caudate and produces increased activity in the head of the caudate. Hypothetically, primitive cleaning and checking behaviors are "hard-wired" in the thalamus. Insel's hypothesis is supported by evidence from MRI (magnetic resonance imaging) studies, which have found an abnormally small caudate in some OCD patients, and by positron emission tomography (PET scan) studies, which have found increased metabolism in orbital frontal cortex, cingulate gyrus, and caudate, with decreases following successful treatment. The association of OCD with Tourette's syndrome and Sydenham's chorea, which are believed to involve basal ganglia pathology, is also consistent with this model. In the section below, we have outlined a more detailed description of the various theories and hypotheses involved in the biological basis of OCD. As will be noted by many readers, the information provided here is more suited for clinicians, medical professionals, or others who are more familiar with medical terminalogy. For this reason, readers are encouraged to seek further information from their physicians/psychiatrists and/or other OCD resources. Etiology: Biological Models Functional Neuroanatomy Many investigators have contributed to the hypothesis that OCD involves dysfunction in a neuronal loop running from the orbital frontal cortex to the cingulate gyrus, striatum (cuadate nucleus and putamen), globus pallidus, thalamus and back to the frontal cortex. Organic insult to these regions can produce obsessive and compulsive symptoms. The results of neurosurgical treatment of OCD strongly support this hypothesis. Surgical interruption of this loop by means of cingulotomy, anterior capsulotomy or subcaudate tractotomy brings about symptomatic improvement in a large proportion of patients unresponsive to all other treatments. Cingulotomy interrupts this loop at the anterior cingulate cortex, thereby disrupting frontal cortical input into the Papez circuit and limbic system, which are believed to mediate anxiety and other emotional symptoms. Anterior capsulotomy (lesions within the anterior limb of the internal capsules) and subcaudate tractotomy (lesions in the substantia innominata, just under the head of the caudate nucleus) interrupt fronto-thalamic fibers, which may mediate the obsessive and compulsive components of OCD. Baxter et al. in 1992 hypothesized that the hyperactivity observed in this neuronal loop arises because of imparied caudate nucleus function. The impariment allows "worry inputs" from the orbitofrontal cortex to inhibit excessively the inhibitory output from the globus pallidus to the thalamus. The resulting excess in thalamic output then impinges on various brain regions involved in the experience of OCD symptoms, including the orbital frontal region, thus reinforcing hyperactivity in the neuronal loop. However, Baxter et al. caution that the abnormal neurophysiology underlying OCD symptoms may involve structures as yet undetected. Decreased metabolic activity can be missed by current scanning techniques. Alternately, the metabolic hyperactivity of small neuronal fields can be missed when surrounding structures exhibit no change or mask the increase behind compensatory decreases. Serotonin and Other Neurotransmitters The hypothesis that an abnormality in serotonergic neurotransmission underlies OCD arose out of the observation that clomipramine, which inhibits both serotonin and norepinephrine reuptake, relieved symptoms, whereas noradrenergic reuptake inhibitors did not. The unique efficacy of clomipramine and the SSRIs remains the strongest support for this hypothesis. Studies of peripheral markers of serotonergic function in blood and cerebrospinal fluid have not conclusively demonstrated an abnormality. Pharmacological challenge studies with serotonergic agonists have suggested dysregulation within the serotonin system, with behavioral hypersensitivity and neuroendocrinological hyposensitivity to the activation of serotonin receptors, but numerous inconsistencies remain to be resolved. The beneficial effects of enhanced serotonergic neurotransmission do not prove that abnormalities in this system are the root cause of OCD symptoms. Serotonergic neurons modulate the function of many other systems, where the primary cause or causes may lie. In patients with comorbid Tourette's syndrome, tics and schizotypal personality disorder, treatment studies indicate a role for dopaminergic neurons. Genetic Contributions Twin studies and family studies strongly suggest that vulnerability to OCD can be inherited, but a positive family history is absent in many patients. Older studies of monozygotic twins show a 65% concordance for OCD, but no control groups were included. One study found an 87% concordance for "obsessional features" (OCD symptoms that may not have caused significant distress or social impairment) in monozygotic twins; the concordance of dizygotic twins was only half as large. On the other hand, none of eight monozygotic twin pairs in another study were concordant for OCD, according to Andrews et al. in 1990. A recent review notes that in Pauls' study in 1992, 10% of the parents of children and adolescents with OCD themselves had the disorder, and in another study, OCD was present in 25% of fathers and 9% of mothers. The symptoms of parents and children usually differed, arguing aginst social or cultural transmission. A study by Black et al. in 1992 however, found no increase in OCD prevalence in first degree relatives of OCD patients compared to those of control group. The recent finding, by Murphy et al. in 1997 and Swedo et al. in 1997, that an antigen which is a genetic marker for rheumatic fever susceptibility is also a marker for susceptibility to an autoimmune form of childhood onset OCD will undoubtedly spur progress in unraveling genetic contributions to the pathogenesis of OCD. Source: OCD Stanford

First day of senior year tomorrow and my anxiety level is through the roof. Please send advice and tips my way that helped you through high school while struggling with OCD and anxiety! It would be very much appreciated.